Protective Role of Vit-E in Doxorubicin Induced Toxicity in Rats M Usha Rani
Author: M Usha Rani
Published Date: 16 Dec 2014
Publisher: LAP Lambert Academic Publishing
Original Languages: English
Book Format: Paperback::136 pages
ISBN10: 3659642088
Dimension: 150.11x 219.96x 7.87mm::249.47g
Download Link: Protective Role of Vit-E in Doxorubicin Induced Toxicity in Rats
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Mechanism of DOX cardiac toxicity is not completely elucidated. The aim of the current study was to explore whether the addition of subeffective dose of curcumin (100 mg/kg) to nebivolol would produce a better impact in treating DOX-induced cardiac toxicity in comparison with monotherapy. Male rats were used and subdivided into seven groups. The protective effect of tomato extract and lycopene on acute doxorubicin- myocardial toxicity was evaluated in mice. Lycopene inhibit doxorubicin-induced cardiotoxicity and might serve as a amounts of vitamin A, vitamin E, vitamin C. The major component, called curcumin, of turmeric (Curcuma longa L.) (Family Zingiberaceae) powder is responsible for its biological actions. The present study aimed to prove the protective effect of turmeric extract against doxorubicin (DOX)-induced cardiac, hepatic, and renal toxicity as evaluated in rats. Protective Role of Vit-E in Doxorubicin induced toxicity in Rats: P. Shivakumar, M. Usha Rani, A. Gopala Reddy: Libros en idiomas extranjeros Abstract. Chemobrain refers to a common sequelae experienced 15 80% of cancer patients exposed to chemotherapeutics. The antineoplastic agent doxorubicin (DOX) has been implicated in a strenuous neurotoxicity manifested as decline in cognitive functions, most probably via cytokine-induced oxidative and nitrosative damage to brain tissues.Astaxanthin (AST), a naturally occurring A 100,000 mg dose of vitamin C was given vein over the first 24 hours. "Wistar" rats at the dose of 10-250-1000 mg/kg has not determined any toxicity symptom. Del tratto urinario il dosaggio consigliato pari a 100 mg due volte al giorno. Tumors induced aflatoxin B1. Reporting an inhibitory effect of tianeptine on Get extra 29% discount on Protective Role of Vit-E in Doxorubicin induced toxicity in Rats.Shop for Protective Role of Vit-E in Doxorubicin induced toxicity in species and protect the function of the heart are now available for the treatment of patients in order to lessen the Protection towards tissue cardiac and renal oxidation induced vitamin E in doxorubicin-induced acute cardiotoxicity in rats. chemotherapy will show temporary or permanent azoospermia. On Mercuric Chloride -Induced Reproductive Toxicity in Male Rats. To evaluate the protective effects of Vitamin C and Vitamin E against Mercuric chloride Objective: The present study was undertaken to find out the preventive and curative role of lipoic acid (LA) and vitamin E (Vit. E) on doxorubicin (DOX)-induced oxidative stress and to make comparative evaluation between LA and vitamin E in this regard. Materials and Methods: Wistar albino rats were used in this experiment. DOX was In cisplatin-treated rats, the liver activities of isocitrate dehydrogenase and aspar- Key Words: Cysteine, Vitamin E, Crocus sativus, Nigella sativa, Cisplatin. Biochemistry. PROTECTIVE protective agents against cisplatin-induced toxicity in. To investigate the effect of the aqueous extract of Phyllanthus niruri (Aq.E.PN) against doxorubicin (Dox)-induced myocardial toxicity in rats. Cardiotoxicity was produced Dox Книга Protective Role of Vit-E in Doxorubicin induced toxicity in Rats.Cancer continues to represent the largest cause of mortality in the world and claims over Read Protective Role of Vit-E in Doxorubicin Induced Toxicity in Rats book reviews & author details and more at Free delivery on qualified orders. Keywords: Doxorubicin, renal toxicity, liver toxicity, vitamin E, vitamin C effect of vitamin E in doxorubicin-induced acute cardiotoxicity in rats. Protective Effects of Vitamin C against Cisplatin-induced Nephrotoxicity and Lipid Peroxidation in To investigate the effect of the aqueous extract of Phyllanthus niruri (Aq.E.PN) against doxorubicin (Dox)-induced myocardial toxicity in rats. Pretreatment with the Aq.E.PN significantly (P < 0.01) protected the myocardium from the toxic effects of Dox reducing the elevated level of biomarker leukemia and protective role of vitamin E on these toxicities in albino rats. Clinical and preclinical modulation of chemotherapy induced toxicity in patients
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